EIKEN諾如病毒(諾瓦克病毒)檢測卡

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主要用途：用于檢測糞便標本中的諾如病毒抗原，以支持諾如病毒感染的診斷。

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諾如病毒(諾瓦克病毒)檢測卡

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諾如病毒(諾瓦克病毒)檢測卡

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
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【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-3室
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比利時魯汶天主教大學(xué)的研究人員*成功的利用小鼠研究人類腫瘤模型，研究結(jié)果發(fā)表在了7月24日的 Cell Report 期刊上。
魯汶天主教大學(xué)實驗和臨床研究中心的 Pierre Sonveaux's 教授帶領(lǐng)及其所帶領(lǐng)的團隊前期對腫瘤細胞的線粒體進行了深入研究，進而成功發(fā)現(xiàn)了一個化學(xué)藥物家族，發(fā)揮藥效時能夠防止腫瘤細胞的轉(zhuǎn)移。他們發(fā)現(xiàn)線粒體這一 “細胞動力工廠”的功能改變時，會促進腫瘤細胞的轉(zhuǎn)移。
博士后研究的 Paolo E. Porporato 和 Pierre Sonveaux's 團隊中其他一些年輕的研究人員深入挖掘了線粒體這一功能的分子機制，zui終發(fā)現(xiàn)是由于線粒體在某些特定情況下，會產(chǎn)生大量的自由基——超氧化物，正是這些超氧化物的過剩導(dǎo)致腫瘤細胞的轉(zhuǎn)移，造成不可挽回的后果。
包括帕金森病和阿爾茨海默氏癥等許多其它人類疾病中，線粒體產(chǎn)生的超氧化物都可以被特殊的抗氧化劑所封閉，例如 MitoTEMPO 。小鼠給藥以及人類腫瘤模型給藥的結(jié)果均闡明了，這些化合物能夠高效地阻斷腫瘤細胞的遷移，并且防止小鼠體內(nèi)人類腫瘤的自發(fā)形成。
這項研究發(fā)現(xiàn)給予了研究人員極大的鼓勵，為今后臨床試驗中腫瘤細胞轉(zhuǎn)移的防治奠定了基礎(chǔ)。

Researchers at the Catholic University of Leuven in Belgium for the first time successfully used mice to study human tumor models, the findings of which were published in the July 24 issue of the Cell Report.
Led by Pierre Sonveaux's professor of the Center for Experimental and Clinical Studies at the Catholic University of Leuven and his team conducted an in-depth study on the mitochondria of tumor cells in advance and successfully discovered a family of chemical drugs that can prevent tumor cell metastasis . They found that when the function of mitochondria, a "cell-powered factory," changed, they promoted tumor cell metastasis.
Paolo E. Porporato, a postdoctoral researcher, and other young researchers at Pierre Sonveaux's team delve into the molecular mechanisms of mitochondrial function, culminating in mitochondria that produce large amounts of free radicals in certain cases Oxides, it is the excess of these superoxides that cause the metastasis of tumor cells, resulting in irreparable consequences.
In many other human diseases, including Parkinson's disease and Alzheimer's disease, the mitochondrial superoxide can be blocked by special antioxidants such as MitoTEMPO. The results of administration to mice as well as administration to human tumor models have demonstrated that these compounds are highly effective in blocking the migration of tumor cells and prevent the spontaneous formation of human tumors in mice.
The study found great encouragement for researchers and laid the foundation for the prevention and treatment of tumor cell metastasis in clinical trials in the future.